| In vitro: |
| Russian Journal of Plant Physiology, 2008, 55(2):193-200. | | Effect of epibrassinolide on tyrosine phosphorylation of the calvin cycle enzymes.[Reference: WebLink] |
METHODS AND RESULTS:
The plants of Brassica juncea (L) were grown in the presence of NaCl and/or NiCl2 and were sprayed with 1muM of 24-Epibrassinolide (EBL) at 15 days after sowing (DAS) and were sampled at 30 DAS.
The plants exposed to NaCl and/or NiCl2 exhibited a significant decline in growth, the level of pigments and photosynthetic parameters. However, the follow up treatment with EBL detoxified the stress generated by NaCl and/or NiCl2 and significantly improved the above parameters. The NaCl and/or NiCl2 increased electrolyte leakage and lipid peroxidation, and decreased the membrane stability index (MSI) and relative water content. However, the EBL treatment in absence of the stress improved the MSI and relative water content but could not influence electrolyte leakage and lipid peroxidation.
CONCLUSIONS:
The antioxidative enzymes and the level of proline exhibited a significant increase in response to EBL as well as to NaCl and/or NiCl2 stress. | | Planta. 1998 Dec;207(1):120-4. | | Effect of epibrassinolide on hypocotyl growth of the tomato mutant diageotropica.[Pubmed: 11536896 ] |
METHODS AND RESULTS:
The effect of Epibrassinolide (epiBR) on the growth and gravitropism of hypocotyls was investigated in diageotropica (dgt), a mutant of tomato (Lycopersicon esculentum Mill.). The elongation of (dgt) hypocotyls, which had been reported to be auxin-insensitive, was promoted by increasing concentrations of indole-3-acetic acid (IAA) in the presence of epiBR. alpha-(p-Chlorophenoxy)isobutyric acid, an inhibitor of auxin action, blocked the enhancement of growth by IAA and epiBR. Time course analysis of IAA-induced dgt hypocotyl elongation in the presence of epiBR revealed typical auxin response kinetics.
CONCLUSIONS:
These results suggest that epiBR restores the auxin responsiveness of dgt hypocotyls with respect to elongation. However, epiBR did not rescue the dgt phenotype with respect to shoot gravitropism. It was therefore concluded that brassino-steroid insensitivity or deficiency is not the primary defect of the dgt mutation. |
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