| Journal of Applied Electrochemistry, 1999, 29(5):593-599. |
| 4-Aminoantipyrine as an inhibitor of mild steel corrosion in HCl solution.[Reference: WebLink] |
METHODS AND RESULTS:
4-aminoantipyrine (AAP) was tested as a corrosion inhibitor for mild steel in 2 M HCl solution using different techniques: weight loss, potentiodynamic polarization and electrochemical impedance spectroscopy (EIS). The results showed that AAP is an inhibitor for mild steel in this medium. The inhibition was assumed to occur via adsorption of the inhibitor molecule on the metal surface. In the 20 to 60 ∘C temperature range, the AAP adsorption follows the Flory–Huggins isotherm and/or the El-Awady et al. kinetic-thermodynamic model.
CONCLUSIONS:
The protection efficiency increases with increasing inhibitor concentration (in the range 10−310−2 M) but decreases with increasing temperature. The thermodynamic functions of dissolution and adsorption processes were calculated.
|
| Journal of hazardous materials, 2011, 192(3):1766-1771. |
| Effect of 4-aminoantipyrine on oxidative stress induced by glutathione depletion in single human erythrocytes using a microfluidic device together with fluorescence imaging.[Reference: WebLink] |
METHODS AND RESULTS:
The effects of 4-aminoantipyrine (AAP) on oxidative stress induced by glutathione (GSH) depletion in single human erythrocytes were investigated using microfluidic technique and fluorescence imaging. Most cell-based toxicity evaluations on GSH are performed with bulk experiments based on analysis of cell populations. This work established a single-cell toxicity evaluation method to statistically analyze the GSH amount in single erythrocytes incubated with AAP in different concentrations. The experimental conditions of cell flow rate and cell concentration were optimized. The GSH contents in erythrocytes decreased with increasing dose of AAP. At low concentration, AAP had a little effect on GSH; while at high concentration, AAP led to GSH depletion reaching a maximum of 14.53%. The depletion of GSH leads to a significant shift to a more oxidizing intracellular environment.
CONCLUSIONS:
This study provides basic data for presenting the effect of AAP on GSH in erythrocytes and is helpful for understanding its toxicity during the blood transportation process. In addition, it will also complement studies on the environmental risk assessment of AAP pollution. |
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